In this view, BHT gets in between the lipids and the hydrophobic regions of the proteins that are normally immersed in lipid. By altering the dynamics between these hydrophobic surfaces, the structure is destabilized and either disintegrates or selective proteins become detached.
In test tube experiments, BHT has not altered the growth of the host cell culture. The mechanism of BHT's activity apparently involves interference in the associations between envelope lipids and hydrophobic regions in envelope proteins. This effect is seen at concentrations of BHT that do not alter mammalian lipid membrane structure. This research presents a potentially powerful technology.
In addition to BHT as a support nutrient for those who have ( ), R. Edward Hope-Simpson used meticulous, and solitary, detective work to discover that the ( ) was reactivated in adults. Dr. Hope-Simpson's work led to Vitamin D's association (or the lack of) to another ( ). Proc R Soc Med. 1965 Jan;58:9-20.
The Clinical Use of BHT
Most of the studies presented here on the effect of BHT (butylated hydroxytoluene, hydroxy toluene) on various organ systems use levels of antioxidant of between 50 mg/kg and 500 mg/kg. While the higher dose is conclusively tied to adverse effects in some animals, the lower dose seems to be quite benign.
For a 70 kg person, 50 mg/kg is a 3500 mg dose, which is 100 times the unconditionally acceptable daily intake of 0.50 mg/kg body weight set by the WHO (Joint FAO/WHO Expert Committee on Food Additives, 1967). Taking 250-2000 mg/day are suggested, which is 7-58 times the WHO unconditionally acceptable dose for a 70 kg person. For a 50 kg person, 250 mg is 10 times the WHO dose.Warnings Information:
(butylated hydroxytoluene, hydroxy toluene) |
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